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From: TSS ()
Subject: Bush Administration May Ease 'Downer Cattle' Ban (a regime run amuck, renegades, lawlessness and there scared cows)
Date: April 15, 2005 at 12:55 pm PST

-------- Original Message --------
Subject: Bush Administration May Ease 'Downer Cattle' Ban
Date: Fri, 15 Apr 2005 14:56:00 -0500
From: "Terry S. Singeltary Sr."
To: Bovine Spongiform Encephalopathy , Gary Burkholder , mike@nobull.net, BillBullard@R-Calfusa.com


Bush Administration May Ease 'Downer Cattle' Ban
Fri Apr 15, 2005 02:55 PM ET


By Randy Fabi

WASHINGTON (Reuters) - The Bush administration said on Friday it may
allow some injured cattle to be slaughtered for human food, easing a
regulation that the Agriculture Department adopted 15 months ago after
the nation's first case of mad cow disease.

Consumer groups said they oppose any changes in regulations aimed at
keeping the deadly disease out of the food supply.

The USDA prohibited all so-called downer cattle -- those too sick or
injured to walk -- from being slaughtered for human food soon after a
Washington state dairy cow was diagnosed with bovine spongiform
encephalopathy (BSE) in December 2003.

The ban was part of a package of tighter USDA regulations to prevent mad
cow disease, whose symptoms can include an inability to walk.

Agriculture Secretary Mike Johanns suggested that the ban on downer
cattle may be eased after the USDA completes an enhanced surveillance
program of U.S. cattle later this year.

"There is a compelling argument: If you've got an animal that's clearly
under 30 months that broke a leg in transit, there is no threat of BSE
whatsoever," Johanns told reporters after addressing the National
Cattlemen's Beef Association.

"Why are we doing this? I'm going to thoughtfully consider those
arguments," he added.

Scientists believe that mad cow disease is spread through contaminated
livestock feed. Young animals are considered to pose the least risk of
disease because BSE takes several years to incubate.

The ban on downer cattle being slaughtered for human food represents a
sizable financial loss to cattle ranchers. For example, a 1,110-pound
steer is worth around $1,000 if slaughtered for steaks and ground beef,
but brings less than $200 if condemned as a downer and used for pet food.

About 195,000 cattle are downers out of more than 30 million slaughtered
annually, according to industry estimates.

USDA officials previously said the department would review all of its
anti-mad cow regulations after it completes an expanded testing program
sometime in 2005. Johanns' comments on Friday were the most explicit to
date of potential changes the government is examining.

"When we get to a point where we're ready to wrap up the increased
surveillance and decide what next to do, I want to look at a range of
issues," said Johanns, a former governor of Nebraska, a major
cattle-producing state. Sen. Ben Nelson, a Nebraska Democrat, will take
part in the review, he said.

No other cases of BSE have been found in the U.S. cattle herd, despite
expanded testing since June 2004.

As of April 10, the USDA tested 314,394 animals in its expanded
surveillance program. That will be completed in the next few months,
opening the door for USDA to propose changes based on its findings.

Consumer advocates said cattle unable to walk should not be used for
human consumption.

"I'm not surprised to hear that the Bush administration might backtrack
on important BSE protections if the surveillance program doesn't turn up
additional positives," said Caroline Smith DeWaal, food safety director
for the Center for Science in the Public Interest.

"Downer cattle represent less-healthy animals and should be kept out of
the food supply," she added.

Other farm groups have expressed concern that the ban on downer cattle
could eventually lead to a similar restriction on pigs sent to slaughter.

The package of mad cow prevention measures adopted by the USDA 15 months
ago included a ban on using brains and small intestines from older
cattle for human food and a ban on stunning cattle with a powerful air
injection to the skull.

The Food and Drug Administration is still considering whether to ban the
use of cattle blood as a protein supplement for calves and the use of
chicken litter as cattle feed.

© Reuters 2005. All Rights Reserved.


http://www.reuters.com/newsArticle.jhtml?type=healthNews&storyID=8198234

http://www.reuters.com/newsArticle.jhtml;jsessionid=TQBYBCTN0HZJWCRBAEKSFEY?type=healthNews&storyID=8198234&pageNumber=1

Greetings,

THIS administration has totally run amuck. There like a band of
renegades with all there sacred cows.
Absolutely and Totally lawless. A band of outlaws. A frightening regime
to fear. ...

> "There is a compelling argument: If you've got an animal that's
> clearly under 30 months that broke a leg in transit, there is no
> threat of BSE whatsoever," Johanns told reporters after addressing the
> National Cattlemen's Beef Association.
>

and without any knowledge of science and TSEs. there just might be a
reason why a cow stumbled, staggered, fell
and broke a leg. IT just might be the first signs of BSE or BaSE ;

Issued: Monday, 28 August 2000
NEW EVIDENCE OF SUB-CLINICAL PRION INFECTION: IMPORTANT RESEARCH
FINDINGS RELEVANT TO CJD AND BSE

A team of researchers led by Professor John Collinge at the Medical
Research Council Prion Unit1 report today in the Proceedings of the
National Academy of Sciences, on new evidence for the existence of a
'sub-clinical' form of BSE in mice which was unknown until now.

The scientists took a closer look at what is known as the 'species
barrier' - the main protective factor which limits the ability of
prions2 to jump from one species to infect another. They found the mice
had a 'sub-clinical' form of disease where they carried high levels of
infectivity but did not develop the clinical disease during their normal
lifespan. The idea that individuals can carry a disease and show no
clinical symptoms is not new. It is commonly seen in conventional
infectious diseases.

Researchers tried to infect laboratory mice with hamster prions3 called
Sc237 and found that the mice showed no apparent signs of disease.
However, on closer inspection they found that the mice had high levels
of mouse prions in their brains. This was surprising because it has
always been assumed that hamster prions could not cause the disease in
mice, even when injected directly into the brain.

In addition the researchers showed that this new sub-clinical infection
could be easily passed on when injected into healthy mice and hamsters.

The height of the species barrier varies widely between different
combinations of animals and also varies with the type or strain of
prions. While some barriers are quite small (for instance BSE easily
infects mice), other combinations of strain and species show a seemingly
impenetrable barrier. Traditionally, the particular barrier studied here
was assumed to be robust.

Professor John Collinge said: "These results have a number of important
implications. They suggest that we should re-think how we measure
species barriers in the laboratory, and that we should not assume that
just because one species appears resistant to a strain of prions they
have been exposed to, that they do not silently carry the infection.
This research raises the possibility, which has been mentioned before,
that apparently healthy cattle could harbour, but never show signs of, BSE.

"This is a timely and unexpected result, increasing what we know about
prion disease. These new findings have important implications for those
researching prion disease, those responsible for preventing infected
material getting into the food chain and for those considering how best
to safeguard health and reduce the risk that theoretically, prion
disease could be contracted through medical and surgical procedures."

ISSUED FRIDAY 25 AUGUST UNDER EMBARGO. PLEASE NOTE THAT THE EMBARGO IS
SET BY THE JOURNAL.

FOR FURTHER INFORMATION CONTACT THE MRC PRESS OFFICE ON 020 7637 6011
(OFFICE HOURS) OR 07818 428297 OR 0385 774357 (OUT-OF-OFFICE-HOURS) OR
PROFESSOR JOHN COLLINGE ON 020 7594 3760. PLEASE NOTE THAT OWING TO
TRAVEL COMMITMENTS PROFESSOR COLLINGE WILL ONLY BE AVAILABLE UNTIL 16.30
ON FRIDAY 25 AUGUST AND CONTACTABLE AGAIN ON MONDAY 28 AUGUST VIA THE
MRC PRESS OFFICE. DR ANDREW HILL (A CO-AUTHOR ON THE PAPER) FROM THE
DEPARTMENT OF PATHOLOGY AT THE UNIVERSITY OF MELBOURNE WILL BE AVAILABLE
ON 00 61 3 8344 3995 (DURING OFFICE HOURS) OR 00 61 3 9443 0009
(OUT-OF-OFFICE HOURS). PLEASE NOTE THAT AUSTRALIA IS TEN HOURS AHEAD OF
UK TIME.

NOTES FOR EDITORS

Professor Collinge is a consultant neurologist and Director of the newly
formed MRC Prion Unit based at The Imperial College School of Medicine
at St Mary's Hospital. He is also a member of the UK Government's
Spongiform Encephalopathy Advisory Committee (SEAC). The MRC prion unit
is was set up in 1999, and its work includes molecular genetic studies
of human prion disease and transgenic modelling of human prion diseases.

Prions are unique infectious agents that cause fatal brain diseases such
as Creutzfeldt-Jakob disease (CJD) in humans and scrapie and BSE (mad
cow disease) in animals. In some circumstances prions from one species
of animals can infect another and it is clear that BSE has done this to
cause the disease variant CJD in the UK and France. It remains unclear
how large an epidemic of variant CJD will occur over the years ahead.

The strain of prion used here to infect the mice is the Sc237 strain
(also known as 263K) which infects hamsters, and until now was assumed
not to infect mice.

This research was funded by the Medical Research Council and Wellcome Trust.

The Medical Research Council (MRC) is a national organisation funded by
the UK tax-payer. Its business is medical research aimed at improving
human health; everyone stands to benefit from the outputs. The research
it supports and the scientists it trains meet the needs of the health
services, the pharmaceutical and other health-related industries and the
academic world. MRC has funded work which has led to some of the most
significant discoveries and achievements in medicine in the UK. About
half of the MRC's expenditure of £345 million is invested in over 50 of
its Institutes and Units, where it employs its own research staff. The
remaining half goes in the form of grant support and training awards to
individuals and teams in universities and medical schools.

The Wellcome Trust is the world's largest medical research charity with
a spend of some £600 million in the current financial year 1999/2000.
The Wellcome Trust supports more than 5,000 researchers, at 400
locations, in 42 different countries to promote and foster research with
the aim of improving human and animal health. As well as funding major
initiatives in the public understanding of science, the Wellcome Trust
is the country's leading supporter of research into the history of medicine.

©2002 Medical Research Council
Data Protection policy | Contact the MRC

http://www.mrc.ac.uk/index/public_interest/public-press_office/public-press_releases_2000/public-mrc-43-00.htm

https://web01.aphis.usda.gov/regpublic.nsf/0/eff9eff1f7c5cf2b87256ecf000df08d?OpenDocument


Following recently published research into Transmittable Spongiform
Encephalopathy
(TSE) in mice, at their 29 September meeting, SEAC discussed the
possibility that prion
disease might exist in sub-clinical form.
The research demonstrates that asymptomatic mice that are not thought to be
susceptible to TSE infection are still able to replicate prion protein
to high levels and
could transmit disease to other mice.
Members noted that existing UK control measures on food safety of beef
products
already took into account the possibility that animals with no clinical
signs may be
incubating disease. SEAC was concerned, however, that in other countries
where BSE
was present and less stringently monitored, sub-clinical infection could
occur.
The committee also recommended further research into pigs and poultry to
confirm
earlier findings that these species appear not to harbour TSE infection
after oral exposure.
Sub-clinical prion disease?

http://www.food.gov.uk/multimedia/pdfs/fsanews2.pdf

Rev Sci Tech. 1992 Jun;11(2):605-34.

Sub-acute, transmissible spongiform encephalopathies: current concepts
and future needs.

Bradley R, Matthews D.

Ministry of Agriculture, Fisheries and Food, Central Veterinary
Laboratory, Addlestone, Surrey, United Kingdom.

The first diagnosis of bovine spongiform encephalopathy (BSE) in the
United Kingdom in 1986 was to stimulate the most intensive
epidemiological study of any animal disease of all time in that country.
It led also to the initiation of a broad-based research programme with
an international flavour. This principally involved scientists and
veterinarians in Europe (especially the United Kingdom) and the United
States of America, especially those with experience of slow infections
in general and experimental scrapie in particular. This final chapter
highlights some of the significant discoveries made in the study of BSE
and related diseases of this group but also emphasises the deficits in
knowledge which need to be corrected before such diseases as scrapie in
sheep and goats can be brought under control. The benefits resultant
upon effective disease control will be manifest as improvement in animal
production, welfare and, importantly, the removal of trading barriers
currently in place to protect countries in which diseases such as BSE
and scrapie do not exist. Of key importance is the development of a
simple, cheap and effective diagnostic test for use in the live animal
before the onset of clinical signs. This will be difficult since the
nature of the causal agents is uncertain and none provokes either a
detectable immune response or inflammatory reaction in the host. The
earlier chapters, written by acknowledged specialists from around the
world, deal with the specific diseases in detail and all present some of
the most recent knowledge available. Here the authors emphasise the
important role that major national and international agencies have in
effecting the highest level of control possible in the absence of key
information. International collaboration with countries in which these
diseases exist, and as well as those where they are absent, is of
paramount importance. It is essential that the BSE epidemic which has
severely affected the cattle industry of the United Kingdom is not
allowed to happen in developing countries. Whereas the former has
implemented stringent control measures based on scientific knowledge and
is well on the way to eradicating the disease, the latter could have
much greater difficulty in establishing control. The answer is clear.
BSE must be prevented from occurring elsewhere. To do that, knowledge of
BSE and other members of the group should be widely dispersed and it is
the purpose of this issue to do just that.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=1617204


NEW findings of BASE in cattle in Italy of Identification of a second
bovine amyloidotic spongiform encephalopathy: Molecular similarities
with sporadic Creutzfeldt-Jakob disease

http://www.pnas.org/cgi/content/abstract/0305777101v1


Adaptation of the bovine spongiform encephalopathy agent to primates and
comparison with Creutzfeldt- Jakob disease: Implications for human
health THE findings from Corinne Ida Lasmézas*, [dagger] , Jean-Guy
Fournier*, Virginie Nouvel*, Hermann Boe*, Domíníque Marcé*, François
Lamoury*, Nicolas Kopp [Dagger ] , Jean-Jacques Hauw§, James Ironside¶,
Moira Bruce [||] , Dominique Dormont*, and Jean-Philippe Deslys* et al,
that The agent responsible for French iatrogenic growth hormone-linked
CJD taken as a control is very different from vCJD but is similar to
that found in one case of sporadic CJD and one sheep scrapie isolate;

http://www.pnas.org/cgi/content/full/041490898v1


Characterization of two distinct prion strains derived from bovine
spongiform encephalopathy transmissions to inbred mice

http://vir.sgmjournals.org/cgi/content/abstract/85/8/2471


TSS





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