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From: TSS (
Subject: THEY ANTICIPATE ALARMING INCREASE OF CASES OF ALZHEIMER IN MEXICO and USA (finding the many potential routes and sources of this agent ?)
Date: December 23, 2004 at 2:39 pm PST

-------- Original Message --------
Date: Wed, 22 Dec 2004 14:51:16 -0600
From: "Terry S. Singeltary Sr."
Reply-To: Bovine Spongiform Encephalopathy

##################### Bovine Spongiform Encephalopathy #####################

- Machine Translation by -
Tuesday 21 of December, 12:49 p.m.
Mérida, Yuc., 21 Dic (Notimex). - At least five percents of the Mexican
population suffer Alzheimer and it is anticipated that the incidence of
the disease will increase of alarming way in next the 30 years, by the
aging of the population, noticed the specialist Raul Mena Lopez.
When giving to know the accomplishment, of the 30 of next January to the
4 of February, of the Course the International on Diagnosis and
Treatment of the Disease of Alzheimer. Of Normality to the Dementia, it
indicated that one of the main problems of the Alzheimer is its
inadequate association to madness and, therefore, the segregation of the
The president of the National Committee for the Protection of the Aging
commented that, according to estimations, at world-wide level until year
2000 at least 18 million people, mainly adult greater, suffered Alzheimer.
At world-wide level, he exposed, 75 percents of the cases concentrates
itself in developed nations, in special of Europe, but esteem that in 30
years the greater volume of people with Alzheimer will be in nations
developing like Mexico.
He added that the nations with greater degree of aging and its
association to problems of mental health are Germany, France and
Belgium, this last country with an aging process that lasted 100 years,
but that in nations as Mexico is of 30 years.
He commented that the lack of qualification in its detection on the part
of the general doctors and the population in general, causes that the
suffering is diagnosed when has a high level of advance, in average
after the 60 years.
By such reason, the mental aging and problems, already consider like a
problem epidemiologist who must be taken care of in Mexico where at the
present time as soon as public policies of attention are created.
He explained that the Alzheimer is considered a disease in which the
patient "is dead in life", and its affection has a high degree of
emotional and depressive impact, not only in the affected person, but
also in who takes care of it.
He declared that that condition also generates a rejection social,
because the patient suffers a catalogued disease of dementia and that
socially she is confused with madness.

Mena Lopez mentioned that the problems are several that the attention of
this disease in the country faces, like the lack of qualification for
its early detection, attention, hospitable infrastructure, as well as
medicine deficiency in the sector health.
In the matter of investigation, it emphasized that it is necessary to
impel regional studies on the disease, because has been verified that
hereditary questions, the atmosphere, climate or the food, can be a
factor that stimulates the disease in a society.
In relation to the international course, it aimed that this one will be
fifth that is developed to Latin American level, reason why the
participation of near 800 participants is expected, between doctors and
specialistic ponentes of all the continent.
Within the framework of the forum also the Advanced training courses
will be made in Manejo of the Depression in the Greater Adult, as well
as the National Course of Qualification for caretakers of patients of
Alzheimer, that will extend to all the country.
The encounter is promoted by organizations like the Pan-American
Organization of the Health, the Mexican Foundation for Salud and
Alzheimer{s Disease Internacional (ADI), among others.

Copyright © 2004 Notimex S.A. de C.V. Todos los derechos reservados.
Copyright © 2004 Yahoo! de México S.A. de C.V. Todos los derechos


Prevelance statistics about Alzheimer's Disease: The following
statistics relate to the prevalence of Alzheimer's Disease:

* 4.5 million cases in America (Alzheimers Association, 2004)
* Prevalence of Alzheimer's expected to increase to 11.3-16 million
cases in America by 2050 (Alzheimers Association, 2004)
* 4 million cases of Alzheimer's in the US (ADEAR)
* 19 million people had a family member with Alzheimer's in the US
* 1 in 10 over 65 years in the US have Alzheimer's (ADEAR)
* Nearly 50% of those over 85 years in the US have Alzheimer's (ADEAR)


WONDER what the cause(S) of Alzheimer's is?

COULD it be a low level/dose TSE?

IF i understand correctly, the case against this would be transmission?

IT has not been _documented_ as an infectious disease.

BUT is it?

CJD1/9 0185

Ref: 1M51A

Dr McGovern,


From: Dr A Wight

5 January 1993

Dr Metters
Dr Skinner
Dr Pickles
Dr Morris
Mr Murray


1. CMO will wish to be aware that a meeting was held ai DH yesterday,
4 January, to discuss the above findings. It was chaired by Professor
Murray (Chairman of the MRC Co-ordinating Committee on Research in
the Spongiform Encephalopathies in Man), and attended by relevant
experts in the fields of Neurology, Neuropathology, molecular biology,
amyloid biochemistry, and the spongiform encephalopathies, and by
representatives of the MRC and AFRC.

Briefly, the meeting agreed that:

i) Dr Ridley et al's findings of experimental induction of p amyloid
in primates were valid, interesting and a significant advance in the
understanding of neurodegeneradve disorders;

ii) there were no immediate implications for the public health, and no
further safeguard; were thought to be necessary at present; and

iii) additional research was desirable, both epidemiological and at the
molecular level. Possible avenues are being followed up by DH
and the MRC, but the details will require further discussion.


>ii) there were no immediate implications for the public health,

r i g h t ....

>iii) additional research was desirable, both epidemiological and at the
>molecular level. Possible avenues are being followed up by DH
>and the MRC, but the details will require further discussion.

i wonder where this research follow-up and other possible avenues they
spoke of could be located at?

WITH Mexico having some 5.5 million and USA at 4.5 million,
seems there should be some concern here as the route and source
of this agent that causes Alzheimer's...

Proof Mad Cow Is The Same
As Alzheimer's And CJD?

How Many Of Them Are Really Mad Cow/vCJD/TSEs ???
How Can Government Claims Of Just 'One In A Million' Be Accurate
When CJD Is Not A Reportable Disease? And When The Elderly Do
Not Get Routinely Autopsied??
By Terry Singletary, Sr

Note - This extensive, powerful assemblage of science was first posted
on 1-24-3. The
following data is even more important today. -ed

Our results correspond partially with a recent
study from the Netherlands where an association
between PRNP codon 129 homozygosity, particularly
for Val, and patients with EOAD was reported.7

7. Dermaut B, Croes E, Rademakers R, et al. PRNP Val129 homozygosity
increases risk for early onset Alzheimers disease. Ann Neurol 2003;53:

Full Text ;

Coexistence of CJD and Alzheimer's disease: An autopsy case showing
typical clinical features of CJD
Kuniaki Tsuchiya1,2,3
Saburo Yagish!ta4
Kenji Ikeda2
Michio Sano5
Kazuhiro Taki6
Keisuke Hashimoto6
Sadakiyo Watabiki3

and Hidehiro Mizusawa7

The present report concerns an autopsy case of CJD showing typical
clinical features of CJD. The patient was a Japanese woman without
hereditary burden or dementing disorder anamnesis who was 70-years-old
at the time of death. She developed gait disturbance at age 68, followed
by memory impairment, visual disturbance, and myoclonus. A neurological
examination approximately 2 months after the disease onset revealed
akinetic mutism, in addition to periodic synchronous discharges on
electroencephalogram. Serial neuroradiological examinations disclosed
progressive atrophy of the brain. She died of bronchopneumonia 25 months
after the disease onset. The brain weighed 560 g (cerebrum 490 g,
brainstem with cerebellum 70 g). Macroscopically, neuropathological
examination showed prominent atrophy of the cerebrum, caudate nucleus,
and cerebellum, in addition to necrosis of the cerebral white matter,
compatible with panencephalopathic CJD. Histologically, there was
neuronal loss with or without spongiform change in the cerebral cortex,
parahippocampal gyrus, amygdala, striatum, pallidum, thalamus, pontine
nucleus, and cerebellar granule cells, in addition to diffuse
synaptic-type prion staining in the cerebrum and cerebellum.
Furthermore, senile plaques, compatible with definite Consortium to
establish a registry for Alzheimer's disease rank Alzheimer's disease,
and neurofibrillary changes of the limbic system, consistent with stage
IV of Braak's classification, were found. Based on these
clinicopathological findings and a review of the published literature,
it is concluded that there were two forms of coexistence of CJD and
Alzheimer's disease in the same patient.


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