From: TSS ()
Subject: Sen. Glenn Hegar, R-Katy SAYS SCREW THE LAW, LET THEM EAT HORSE
Date: May 12, 2007 at 7:45 am PST
May 11, 2007, 7:11PM
Senate bill's amendment would steer around horse slaughter ban
By ANABELLE GARAY Associated Press Writer
© 2007 The Associated Press
DALLAS — A change quietly tucked into a state Senate bill approved this week seeks to sidestep a 58-year-old Texas law preventing the slaughter of horses for consumption of their meat.
Sen. Glenn Hegar, R-Katy, inserted an amendment into a bill on the duties and regulations of the Texas Animal Health Commission. It said animals tested by the commission would be exempt from the part of the state agriculture code banning the sale of horse meat for human consumption.
Hegar contends allowing horse meat processing in Texas can keep animals from enduring a harsher fate of being abused, neglected or shipped to Mexico.
"We're really just kind of turning our back on the problem by saying, 'Well, if they're not processed here nothing happens to them anymore,' and that's not the case," he said Friday.
However, opponents accuse the plants of cruelly killing young horses and condemn the processing operations as slaughter of an American symbol.
"There's no indication these horses are going to be sent to Mexico. My understanding is since the early 1990s, we slaughtered around 380,000 horses, and now demand has been reduced to about 100,000 a year. We went from 14 plants down to three," said Jill Buckley, senior director of legislative services for the American Society for the Prevention of Cruelty to Animals.
The Senate approved the bill in Austin on Wednesday, and it was sent to the House's agriculture and livestock committee. House members passed a similar bill in the House, but it does not include provisions on horse slaughter.
"That debate will continue throughout the session. We've got several weeks left," Hegar said. "We'll be able to have that discussion and probably still have it again probably next session and through the interim. Who knows when we'll have final conclusion on this issue."
Efforts to continue horse slaughter the United States suffered recent losses in the federal courts and in Congress.
Earlier this year, a federal appeals court effectively shut down two Texas plants that slaughtered horses and exported the meat overseas — Dallas Crown Inc. in Kaufman and Beltex Corp. in Fort Worth. The 5th U.S. Circuit Court of Appeals in New Orleans overturned a lower court ruling that said a 1949 Texas law banning horse slaughter for the sale of meat for food was invalid.
A third plant run by Cavel International Inc. in DeKalb, Ill., is not affected by the ruling. The three facilities are foreign-owned.
The Texas Animal Health Commission tests thousands of horses for diseases each year. But most of those tested were not headed to the state's slaughter houses.
Congress overwhelmingly voted in favor of a bill that stripped funding for horse meat inspections. But the U.S. Department of Agriculture devised a plan to provide the inspections for a fee for slaughter plants. A federal district judge in Washington found the USDA did not follow federal procedures for setting up the inspection fee program and ended it.
http://www.chron.com/disp/story.mpl/ap/tx/4797528.html
Greetings,
IT seems politicians like Sen. Glenn Hegar, R-Katy, it seems they just think they are above the law.
IT does not matter that A federal appeals court has ruled that horse slaughter is illegal in Texas.
http://www.usaha.org/news-alerts.shtml#NEWS2
http://www.elpasotimes.com/breakingnews/ci_5053089
Hegar just side steps the law. With senators like Hegar, who needs crooks. I am sick and tired of
these crooked senators sneaking in bills, and then hiding behind some stupid lie. horses are abused
all the time, does not mean we have to eat them. the triple sss policy of shoot, shovel, and shut the
heck up has been alive and well in Texas for decades, that's why there not finding anymore mad cows,
they simply bury them ;
ceverett@tahc.state.tx.us
For Immediate Release--
Anthrax Confirmed in Sutton County, Texas
Anthrax Confirmed in Sutton County, Texas
snip...
“Anthrax is under-reported, because many ranchers in this area automatically dispose of carcasses and vaccinate livestock when they find dead animals that are bloated or bloody--common signs of the disease,” said Dr. Fancher. “Anthrax is a reportable disease, however, and it’s important to know when an outbreak occurs, so other ranchers can be notified to vaccinate.........
snip...end
"Sen. Glenn Hegar, R-Katy, inserted an amendment into a bill on the duties and regulations of the Texas Animal Health Commission. It said animals tested by the commission would be exempt from the part of the state agriculture code banning the sale of horse meat for human consumption."
SO, does this mean they are going to test these horses for BSE, BASE, and or any strain of TSE ???
IN the USA, the feeding of ruminant protein to horses is a common practice, and don't think for a minute
that horses not susceptible to a TSE, that's just another myth by the USDA ;
IN CONFIDENCE
Suspect BSE in Horse
The Parliamentary Secretary (Mr Maclean) will wish to be aware that, in
making his differential diagnosis, a veterinary surgeon in the Reading area
has included the possibility of BSE in a horse under his care. Athough it is
unlikely to be BSE, because of the symptoms exhbibited the veterinarian
believes that he cannot exclude the possibility. The case was brought to the
notice of one of the veterinary staff at the CVL by the owner's veterinary
surgeon and liaison is being maintained.
The horse in question is a five-year old eventing gelding which was
purchased by the present owner about four months ago. Approximately two
months after purchase the animal became a little apprehensive, developed
mild nervous symptoms and became over-sensitive to noise. The nervous
symptoms have increased and the horse is now practically impossible to ride.
Investigations by the owner's private veterinary surgeon are continuing but
it is likely that the animal will have to be destroyed.
If the horse should die or be destroyed, a full post-mortem examination
will be required for insurance purposes and will probably be carried out at
a non-Ministry laboratory. However, Mr. Bradley of the Pathology Department,
CVL, has informed the private veterinary surgeon that he is willing to
provide a second opinion on the brain histology if requested.
I will keep the Parliamentary Secretary informed of any further
developments in the case.
I CRAWFORD
14 May 1990
Mr. M P H Hill, PS/Parliamentary Secretary
(Mr Maclean) - by FAX
cc: Private Offices - by FAX
Mr. K C Meldrum
Mrs E A J Attridge - by FAX
Mr. D J Evans
Mr. K C Taylor
Mr. R Lowson
Mr. R Bradley, CVL
(hand written notes to follow, hard to read...tss)
The Parliamentary Secretary (Mr. Maclean) was grateful for this. He said
that we must keep very close ........on it, and when the horse dies, or put
down we must be told immediately. He also feels it is very important ... our
veterinary staff are included in the brain examination...........(the rest
cut out......tss)
90/05.14/10.1
http://www.bseinquiry.gov.uk/files/yb/1990/05/14010001.pdf
http://www.bseinquiry.gov.uk/files/yb/1990/06/26009001.pdf
http://www.bseinquiry.gov.uk/files/yb/1990/06/26010001.pdf
36. PMSG was used initially. This was followed by Porcine and Ovine FSH
with a small amount of Equine FSH, (HAP which is equine FSH, which is derived
from horse pituitaries). Human Menopausal Gonodotrophin (HMG), derived from
urine was used following work done by DeLauria and others in Italy largely
funded by Serono but because the demand in the human fertility field and
the expense and inconvenience of the packaging (small vials) was curtailed. It
has been launched in the last few years as a veterinary product under the name
Pergovet but is still very expensive and is reserved for occasional use.
37. As far as LH (Luteinising hormone) is concerned, as I have mentioned
earlier this has been used for the last forty years as a holding injection.
snip...
Paragraphs 3.3, 3.4 & 3.5 of Dr Maddocks’ original statement (WS No 467)
28. (bGH) (bovine growth hormone) was used for a short time in the USA. It
was never allowed in the UK not even the recombinant form is allowed, as it is
in the USA. No incidence of BSE was reported in the USA and they surely would now
be looking for it?
29. Pituitary FSH from pigs has been used in the USA prior to its use in
the UK and much more extensively there and Canada. There is no reported incidence of
BSE in either country.
30. Thousands of embryos were exported from this country to the USA prior
to the ban being imposed. No cases of BSE have occurred in the resultant progeny
or the surrogate mothers.
31. In the UK, two ET companies looked into and did use Porcine FSH
collected from slaughter houses and prepared in house by very experienced and qualified
individuals. In the second case an outside laboratory of good repute did
the extraction.
32. It was most definitely not a case of being a “cottage industry”. This
FSH was not sold to veterinarians or farmers but was distributed in a limited way to
those vets involved in ET.
http://www.bseinquiry.gov.uk/files/ws/s537.pdf
The Premarin Menopause Drug is Made of Horse Urine
http://www.hihopes.com/premarin_info_p2.html
http://www.findings.net/supremarin.html
Science 6 October 2006:
Vol. 314. no. 5796, pp. 133 - 136
DOI: 10.1126/science.1132661
Prev | Table of Contents | Next
Reports
Infectious Prions in the Saliva and Blood of Deer with Chronic Wasting Disease
Candace K. Mathiason,1 Jenny G. Powers,3 Sallie J. Dahmes,4 David A. Osborn,5 Karl V. Miller,5 Robert J. Warren,5 Gary L. Mason,1 Sheila A. Hays,1 Jeanette Hayes-Klug,1 Davis M. Seelig,1 Margaret A. Wild,3 Lisa L. Wolfe,6 Terry R. Spraker,1,2 Michael W. Miller,6 Christina J. Sigurdson,1 Glenn C. Telling,7 Edward A. Hoover1*
A critical concern in the transmission of prion diseases, including chronic wasting disease (CWD) of cervids, is the potential presence of prions in body fluids. To address this issue directly, we exposed cohorts of CWD-naïve deer to saliva, blood, or urine and feces from CWD-positive deer. We found infectious prions capable of transmitting CWD in saliva (by the oral route) and in blood (by transfusion). The results help to explain the facile transmission of CWD among cervids and prompt caution concerning contact with __body fluids__ in prion infections.
1 Department of Microbiology, Immunology, and Pathology, College of Veterinary Medicine and Biological Sciences (CVMBS), Colorado State University (CSU), Fort Collins, CO 80523, USA.
2 Veterinary Diagnostic Laboratory, College of Veterinary Medicine and Biological Sciences (CVMBS), Colorado State University (CSU), Fort Collins, CO 80523, USA.
3 Biological Resource Management Division, National Park Service, Fort Collins, CO 80525, USA.
4 Wildlife Artist Supply Company (WASCO) Inc., Monroe, GA 30655, USA.
5 Warnell School of Forestry and Natural Resources, University of Georgia, Athens, GA 30609, USA.
6 Wildlife Research Center, Colorado Division of Wildlife, Fort Collins, CO 80526, USA.
7 Department of Microbiology, Immunology and Molecular Genetics, University of Kentucky, Lexington, KY 40536, USA.
* To whom correspondence should be addressed. E-mail: edward.hoover@colostate.edu
http://www.sciencemag.org/cgi/content/abstract/314/5796/133
PLEASE NOTE, THESE ARE JUST A FEW MAD HORSE FEED RECALL, not enough room
to list all. ...TSS
CVM Update
January 10, 2001
UPDATE ON RUMINANT FEED (BSE) ENFORCEMENT ACTIVITIES
http://www.fda.gov/cvm/CVM_Updates/bseup.htm
RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINE - CLASS
II_______________________
PRODUCT
Red Cell, Iron Rich Homogenized, Yucca Flavored Vitamin-Iron-Mineral
Supplement for all classes of horses. For Animal Use Only. NET
CONTENTS: 1 GALLON. HORSE HEALTH Products, A Division
of Farnam Companies, Inc. PO Box 34820, Phoenix AZ 85067-4820,
Recall # V-002-2.
Redglo, EQUICARE (brand), Homogenized Energy Building Liquid Multi-
Vitamin Supplement for Horses. EQUICARE PRODUCTS, A
Division of Farnam Companies, Inc., PO Box 34820, Phoenix, AZ,
Recall # V-003-2.
CODE
All codes.
RECALLING FIRM/MANUFACTURER
Farnam Companies, Inc., Phoenix, Arizona, sent a recall letter dated
March 8, 2001, to all distributors via regular first class mail. Firm
initiated recall is ongoing.
REASON
The products contain protein material derived from
bovine mammalian tissues; however, the bags are not labeled with the
required BSE cautionary statement.
VOLUME OF PRODUCT IN COMMERCE
14,000 to 15,000 gallons.
DISTRIBUTION
Nationwide.
http://www.fda.gov/bbs/topics/ENFORCE/2001/ENF00719.html
Non-Ruminant Custom Mix Feeds:
V-393-1 "40% Poultry Feed" manufactured with Buckeye 40%
Poultry Concentrate Crumbles, Item 12100
V-394-1 "40% Hog Feed" manufactured with Buckeye 40% Gro'Em
Lean, Item 20550
V-395-1 "Horse Premium Mixer" manufactured with Buckeye 32%
Premium Mixer Pellets, Item 38000
Code: All bulk custom mix feeds manufactured prior to April 20, 2001.
The customer invoices indicate the type of Buckeye supplement used in the
bulk feed.
REASON:
The bulk custom mix feeds were prepared with ruminant feed supplements
recalled by Buckeye Nutrition due to contamination with protein derived
from mammalian tissues. The non-ruminant bulk custom mix feeds were not
labeled with the required BSE caution statement "Do Not Feed to Cattle or
Other Ruminants."
MANUFACTURER/RECALLING FIRM:
Ferrin Cooperative Equity Exchange, Inc., Carlyle, Illinois
RECALLED BY:
The firm , by letter beginning on June 28, 2001.
FIRM INITIATED RECALL:
Ongoing.
DISTRIBUTION:
IL
QUANTITY:
169 tons of ruminant feeds and 27 tons of non-ruminant feeds
END OF ENFORCEMENT REPORT FOR October 10,
2001.
####
http://www.fda.gov/bbs/topics/ENFORCE/2001/ENF00714.html
RECALL NUMBER, PRODUCT AND CODE:
Recall # Product
V-397-1 Hyland Floating Fishfood, in 50 pound bags
V-398-1 Endurance Plus Extrude Horse Feed, in 50 pound bags
V-399-1 Seminole Ultra Bloom Horse Feed, in 50 pound bags
V-400-1 Wheat Flakes, extruded product in bulk, not bagged
V-401-1 Corn Flakes, extruded product in bulk, not bagged
V-402-1 Capt. Crunch, extruded product in bulk, not bagged
V-403-1 Green Corn Puffs, extruded product in bulk, not bagged
V-404-1 Orange Corn Puffs, extruded product in bulk, not
bagged
V-405-1 Whole Kernel Corn, in 50 pound bags, unlabeled
V-406-1 Soybean Meal, in bulk, not bagged, unlabeled
ALL CODES
REASON:
The animal feed products may contain proteins derived from mammalian
tissues.
The products are not labeled with the required BSE caution statement "Do
Not
Feed to Cattle or Other Ruminants."
MANUFACTURER/RECALLING FIRM:
The Hyland Company, Ashland, Kentucky
RECALLED BY:
Manufacturer, by telephone on July 25, 2001, and letters on July 31, 2001.
FIRM INITIATED RECALL:
Complete
DISTRIBUTION:
KY, GA, NC, FL WV
QUANTITY:
568 tons
END OF ENFORCEMENT REPORT FOR August 29, 2001.
####
http://www.fda.gov/bbs/topics/ENFORCE/2001/ENF00708.html
reminds me of the hound study ;
GAH WELLS (very important statement here...TSS)
HOUND STUDY
AS implied in the Inset 25 we must not _ASSUME_ that
transmission of BSE to other species will invariably
present pathology typical of a scrapie-like disease.
snip...
http://www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf
76 pages on hound study;
http://www.bseinquiry.gov.uk/files/sc/seac16/tab04.pdf
> I thought that in Britain dogs had contracted BSE, but perhaps not.
not so fast here;
The spongiform changes were not pathognomonic (ie.
conclusive proof) for prion disease, as they were atypical,
being largely present in white matter rather than grey matter in
the brain and spinal cord. However, Tony Scott, then head of
electron microscopy work on TSEs, had no doubt that these
SAFs were genuine and that these hounds therefore must have
had a scrapie-like disease. I reviewed all the sections
myself (original notes appended) and although the pathology
was not typical, I could not exclude the possibility that this was
a scrapie-like disorder, as white matter vacuolation is seen
in TSEs and Wallerian degeneration was also present in the
white matter of the hounds, another feature of scrapie.
38.I reviewed the literature on hound neuropathology, and
discovered that micrographs and descriptive neuropathology from
papers on 'hound ataxia' mirrored those in material from
Robert Higgins' hound survey. Dr Tony Palmer (Cambridge) had
done much of this work, and I obtained original sections
from hound ataxia cases from him. This enabled me provisionally to
conclude that Robert Higgins had in all probability detected
hound ataxia, but also that hound ataxia itself was possibly a
TSE. Gerald Wells confirmed in 'blind' examination of single
restricted microscopic fields that there was no distinction
between the white matter vacuolation present in BSE and
scrapie cases, and that occurring in hound ataxia and the hound
survey cases.
39.Hound ataxia had reportedly been occurring since the 1930's,
and a known risk factor for its development was the feeding
to hounds of downer cows, and particularly bovine offal.
Circumstantial evidence suggests that bovine offal may also be
causal in FSE, and TME in mink. Despite the inconclusive
nature of the neuropathology, it was clearly evident that this
putative canine spongiform encephalopathy merited further
investigation.
40.The inconclusive results in hounds were never confirmed,
nor was the link with hound ataxia pursued. I telephoned Robert
Higgins six years after he first sent the slides to CVL.
I was informed that despite his submitting a yearly report to the
CVO including the suggestion that the hound work be continued,
no further work had been done since 1991. This was
surprising, to say the very least.
41.The hound work could have provided valuable evidence
that a scrapie-like agent may have been present in cattle offal long
before the BSE epidemic was recognised. The MAFF hound
survey remains unpublished.
Histopathological support to various other published
MAFF experiments
42.These included neuropathological examination of material
from experiments studying the attempted transmission of BSE to
chickens and pigs (CVL 1991) and to mice (RVC 1994).
http://www.bseinquiry.gov.uk/witness/htm/stat067.htm
It was thought likely that at least some, and probably all, of the cases
in zoo animals were caused by the BSE agent. Strong support for this
hypothesis came from the findings of Bruce and others (1994)
( Bruce, M.E., Chree, A., McConnell, I., Foster, J., Pearson, G. &
Fraser, H. (1994) Transmission of bovine spongiform encephalopathy and
scrapie to mice: strain variation and species barrier. Philosophical
Transactions of the Royal Society B 343, 405-411: J/PTRSL/343/405
), who demonstrated that the pattern of variation in incubation period
and lesion profile in six strains of mice inoculated with brain
homogenates from an affected kudu and the nyala, was similar to that
seen when this panel of mouse strains was inoculated with brain from
cattle with BSE. The affected zoo bovids were all from herds that were
exposed to feeds that were likely to have contained contaminated
ruminant-derived protein and the zoo felids had been exposed, if only
occasionally in some cases, to tissues from cattle unfit for human
consumption.
snip...
http://www.bseinquiry.gov.uk/files/ws/s324.pdf
DEFRA
Department for Environment,
Food & Rural Affairs
Area 307, London, SW1P 4PQ
Telephone: 0207 904 6000
Direct line: 0207 904 6287
E-mail: h.mcdonagh.defra.gsi.gov.uk
GTN:
FAX:
Mr T S Singeltary
P.O. Box 42
Bacliff
Texas
USA 77518
21 November 2001
Dear Mr Singeltary TSE IN HOUNDS
Thank you for e-mail regarding the hounds survey. I am sorry for the long
delay in responding.
As you note, the hound survey remains unpublished. However the Spongiform
Encephalopathy Advisory Committee (SEAC), the UK Government's independent
Advisory Committee on all aspects related to BSE-like disease, gave the
hound study detailed consideration at their meeting in January 1994. As a
summary of this meeting published in the BSE inquiry noted, the Committee
were clearly concerned about the work that had been carried out, concluding
that there had clearly been problems with it, particularly the control on
the histology, and that it was more or less inconclusive. However was agreed
that there should be a re-evaluation of the pathological material in the
study.
Later, at their meeting in June 95, The Committee re-evaluated the hound
study to see if any useful results could be gained from it. The Chairman
concluded that there were varying opinions within the Committee on further
work. It did not suggest any further transmission studies and thought that
the lack of clinical data was a major weakness.
Overall, it is clear that SEAC had major concerns about the survey as
conducted. As a result it is likely that the authors felt that it would not
stand up to r~eer review and hence it was never published. As noted above,
and in the detailed minutes of the SEAC meeting in June 95, SEAC considered
whether additional work should be performed to examine dogs for evidence of
TSE infection. Although the Committee had mixed views about the merits of
conducting further work, the Chairman noted that when the Southwood
Committee made their recommendation to complete an assessment of possible
spongiform disease in dogs, no TSEs had been identified in other species and
hence dogs were perceived as a high risk population and worthy of study.
However subsequent to the original recommendation, made in 1990, a number of
other species had been identified with TSE ( e.g. cats) so a study in hounds
was less
critical. For more details see-
http://www.bseinquiry.gov.uk/files/yb/1995/06/21005001.pdf
As this study remains unpublished, my understanding is that the ownership of
the data essentially remains with the original researchers. Thus
unfortunately, I am unable to help with your request to supply information
on the hound survey directly. My only suggestion is that you contact one of
the researchers originally involved in the project, such as Gerald Wells. He
can be contacted at the following address.
Dr Gerald Wells, Veterinary Laboratories Agency, New Haw, Addlestone,
Surrey, KT 15 3NB, UK
You may also wish to be aware that since November 1994 all suspected cases
of spongiform encephalopathy in animals and poultry were made notifiable.
Hence since that date there has been a requirement for vets to report any
suspect SE in dogs for further investigation. To date there has never been
positive identification of a TSE in a dog.
I hope this is helpful
Yours sincerely 4
HUGH MCDONAGH
BSE CORRESPONDENCE SECTION
================================
flounder wrote:
ill bet they will be clean as a whistle too, considering the notice before
there arrival, what a hoot. what about all that mad cow protein in commerce
in 2007 alone? Over 10 Million pounds went out into commerce just a few
months ago. and 2006 was a banner year for banned ruminant feed in commerce
in the USA. so will they test these cows for export to Japan. Creekstone has
been wanting to test, but USDA has been afraid of what they will find.
wonder if Creekstone with test for USA consumer? seems US government has
stopped testing to find after those last two atypical BSE cows, the one in
Alabama and the one in Texas. not to forget the other mad cow from Texas,
from which it finally took an act of Congress to get it confirmed 7+ months
after a postive rapid test. and not to forget the other Texas highly suspect
stumbling and staggering mad cow they did not test at all, nope, just sent
it straight to be rendered. all in all, the USA surveillance and testing for
TSE in the USA bovine has failed terribly, and Japan will be exporting these
products. THE USA consumer is force fed this junk, Japan has a choice. Good
luck. dont believe me, see for yourself;
10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. MBM IN COMMERCE USA
2007
Date: March 21, 2007 at 2:27 pm PST
RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II
___________________________________
PRODUCT
Bulk cattle feed made with recalled Darling’s 85% Blood Meal, Flash Dried,
Recall # V-024-2007
CODE
Cattle feed delivered between 01/12/2007 and 01/26/2007
RECALLING FIRM/MANUFACTURER
Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.
Firm initiated recall is ongoing.
REASON
Blood meal used to make cattle feed was recalled because it was
cross-contaminated with prohibited bovine meat and bone meal that had been
manufactured on common equipment and labeling did not bear cautionary BSE
statement.
VOLUME OF PRODUCT IN COMMERCE
42,090 lbs.
DISTRIBUTION
WI
___________________________________
PRODUCT
Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL
Prot-Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal,
TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY
Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST
POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI – 8# SPECIAL
DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J – PROTEIN/LACTATION, ROCK
CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC
MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY,
A-BYPASS ML W/SMARTA, Recall # V-025-2007
CODE
The firm does not utilize a code - only shipping documentation with
commodity and weights identified.
RECALLING FIRM/MANUFACTURER
Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm
initiated recall is complete.
REASON
Products manufactured from bulk feed containing blood meal that was cross
contaminated with prohibited meat and bone meal and the labeling did not
bear cautionary BSE statement.
VOLUME OF PRODUCT IN COMMERCE
9,997,976 lbs.
DISTRIBUTION
ID and NV
END OF ENFORCEMENT REPORT FOR MARCH 21, 2007
http://www.fda.gov/bbs/topics/enforce/2007/ENF00996.html
USA MAD COW STRAIN MORE VIRULENT TO HUMANS THAN UK STRAIN
18 January 2007 - Draft minutes of the SEAC 95 meeting (426 KB) held on 7
December 2006 are now available.
snip...
64. A member noted that at the recent Neuroprion meeting, a study was
presented showing that in transgenic mice BSE passaged in sheep may be more
virulent and infectious to a wider range of species than bovine derived BSE.
Other work presented suggested that BSE and bovine amyloidotic spongiform
encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the
prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A
MUTATION FOUND IN CASES OF SPORADIC CJD.
snip...
http://www.seac.gov.uk/minutes/95.pdf
3:30 Transmission of the Italian Atypical BSE (BASE) in Humanized Mouse
Models Qingzhong Kong, Ph.D., Assistant Professor, Pathology, Case Western
Reserve
University
Bovine Amyloid Spongiform Encephalopathy (BASE) is an atypical BSE strain
discovered recently in Italy, and similar or different atypical BSE cases
were also reported in other countries. The infectivity and phenotypes of
these atypical BSE strains in humans are unknown. In collaboration with
Pierluigi Gambetti, as well as Maria Caramelli and her co-workers, we have
inoculated transgenic mice expressing human prion protein with brain
homogenates from BASE or BSE infected cattle. Our data shows that about half
of the BASE-inoculated mice became infected with an average incubation time
of about 19 months; in contrast, none of the BSE-inoculated mice appear to
be infected after more than 2 years.
***These results indicate that BASE is transmissible to humans and suggest
that BASE is more virulent than
classical BSE in humans.***
6:30 Close of Day One
http://www.healthtech.com/2007/tse/day1.asp
SEE STEADY INCREASE IN SPORADIC CJD IN THE USA FROM
1997 TO 2006. SPORADIC CJD CASES TRIPLED, with phenotype
of 'UNKNOWN' strain growing. ...
http://www.cjdsurveillance.com/resources-casereport.html
There is a growing number of human CJD cases, and they were presented last
week in San Francisco by Luigi Gambatti(?) from his CJD surveillance
collection.
He estimates that it may be up to 14 or 15 persons which display selectively
SPRPSC and practically no detected RPRPSC proteins.
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf
THE USDA JUNE 2004 ENHANCED BSE SURVEILLANCE PROGRAM WAS TERRIBLY FLAWED ;
CDC DR. PAUL BROWN TSE EXPERT COMMENTS 2006
The U.S. Department of Agriculture was quick to assure the public earlier
this week that the third case of mad cow disease did not pose a risk to
them, but what federal officials have not acknowledged is that this latest
case indicates the deadly disease has been circulating in U.S. herds for at
least a decade.
The second case, which was detected last year in a Texas cow and which USDA
officials were reluctant to verify, was approximately 12 years old.
These two cases (the latest was detected in an Alabama cow) present a
picture of the disease having been here for 10 years or so, since it is
thought that cows usually contract the disease from contaminated feed they
consume as calves. The concern is that humans can contract a fatal,
incurable, brain-wasting illness from consuming beef products contaminated
with the mad cow pathogen.
"The fact the Texas cow showed up fairly clearly implied the existence of
other undetected cases," Dr. Paul Brown, former medical director of the
National Institutes of Health's Laboratory for Central Nervous System
Studies and an expert on mad cow-like diseases, told United Press
International. "The question was, 'How many?' and we still can't answer
that."
Brown, who is preparing a scientific paper based on the latest two mad cow
cases to estimate the maximum number of infected cows that occurred in the
United States, said he has "absolutely no confidence in USDA tests before
one year ago" because of the agency's reluctance to retest the Texas cow
that initially tested positive.
USDA officials finally retested the cow and confirmed it was infected seven
months later, but only at the insistence of the agency's inspector general.
"Everything they did on the Texas cow makes everything USDA did before 2005
suspect," Brown said. ...snip...end
http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r
CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ...
Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central
Nervous System ... Address for correspondence: Paul Brown, Building 36, Room
4A-05, ...
http://www.cdc.gov/ncidod/eid/vol7no1/brown.htm
PAUL BROWN COMMENT TO ME ON THIS ISSUE
Tuesday, September 12, 2006 11:10 AM
"Actually, Terry, I have been critical of the USDA handling of the mad cow
issue for some years, and with Linda Detwiler and others sent lengthy
detailed critiques and recommendations to both the USDA and the Canadian
Food Agency."
OR, what the Honorable Phyllis Fong of the OIG found ;
Audit Report
Animal and Plant Health Inspection Service
Bovine Spongiform Encephalopathy (BSE) Surveillance Program – Phase II
and
Food Safety and Inspection Service
Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat
Recovery Products - Phase III
Report No. 50601-10-KC January 2006
Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle
Still Remain
http://www.usda.gov/oig/webdocs/50601-10-KC.pdf
EXPORTATION AND IMPORTATION OF ANIMALS AND ANIMAL PRODUCTS:
BSE; MRR AND IMPORTATION OF COMMODITIES, 65758-65759 [E6-19042]
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&T=0&P=3854
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0611&L=sanet-mg&T=0&P=3381
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=498
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0702&L=sanet-mg&T=0&P=10277
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&T=0&P=9972
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=4492
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=2583
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&T=0&P=2470
Diagnosis and Reporting of Creutzfeldt-Jakob Disease
Singeltary, Sr et al. JAMA.2001; 285: 733-734.
http://jama.ama-assn.org/http://www.neurology.org/cgi/eletters/60/2/176#535
BRITISH MEDICAL JOURNAL
BMJ
http://www.bmj.com/cgi/eletters/319/7220/1312/b#EL2
BMJ
http://www.bmj.com/cgi/eletters/320/7226/8/b#EL1
Re: RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob
disease in the United States
Email Terry S. Singeltary:
flounder@wt.net
I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to
comment on the CDC's attempts to monitor the occurrence of emerging
forms of CJD. Asante, Collinge et al [1] have reported that BSE
transmission to the 129-methionine genotype can lead to an alternate
phenotype that is indistinguishable from type 2 PrPSc, the commonest
sporadic CJD. However, CJD and all human TSEs are not reportable
nationally. CJD and all human TSEs must be made reportable in every
state and internationally. I hope that the CDC does not continue to
expect us to still believe that the 85%+ of all CJD cases which are
sporadic are all spontaneous, without route/source. We have many TSEs in
the USA in both animal and man. CWD in deer/elk is spreading rapidly and
CWD does transmit to mink, ferret, cattle, and squirrel monkey by
intracerebral inoculation. With the known incubation periods in other
TSEs, oral transmission studies of CWD may take much longer. Every
victim/family of CJD/TSEs should be asked about route and source of this
agent. To prolong this will only spread the agent and needlessly expose
others. In light of the findings of Asante and Collinge et al, there
should be drastic measures to safeguard the medical and surgical arena
from sporadic CJDs and all human TSEs. I only ponder how many sporadic
CJDs in the USA are type 2 PrPSc?
http://www.neurology.org/cgi/eletters/60/2/176#535
LANCET INFECTIOUS DISEASE JOURNAL
Volume 3, Number 8 01 August 2003
Newsdesk
Tracking spongiform encephalopathies in North America
Xavier Bosch
My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost
my mom to hvCJD (Heidenhain variant CJD) and have been searching for
answers ever since. What I have found is that we have not been told the
truth. CWD in deer and elk is a small portion of a much bigger problem.
49-year-old Singeltary is one of a number of people who have remained
largely unsatisfied after being told that a close relative died from a
rapidly progressive dementia compatible with spontaneous
Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of
documents on transmissible spongiform encephalopathies (TSE) and
realised that if Britons could get variant CJD from bovine spongiform
encephalopathy (BSE), Americans might get a similar disorder from
chronic wasting disease (CWD) the relative of mad cow disease seen among
deer and elk in the USA. Although his feverish search did not lead him
to the smoking gun linking CWD to a similar disease in North American
people, it did uncover a largely disappointing situation.
Singeltary was greatly demoralised at the few attempts to monitor the
occurrence of CJD and CWD in the USA. Only a few states have made CJD
reportable. Human and animal TSEs should be reportable nationwide and
internationally, he complained in a letter to the Journal of the
American Medical Association (JAMA 2003; 285: 733). I hope that the CDC
does not continue to expect us to still believe that the 85% plus of all
CJD cases which are sporadic are all spontaneous, without route or source.
Until recently, CWD was thought to be confined to the wild in a small
region in Colorado. But since early 2002, it has been reported in other
areas, including Wisconsin, South Dakota, and the Canadian province of
Saskatchewan. Indeed, the occurrence of CWD in states that were not
endemic previously increased concern about a widespread outbreak and
possible transmission to people and cattle.
To date, experimental studies have proven that the CWD agent can be
transmitted to cattle by intracerebral inoculation and that it can cross
the mucous membranes of the digestive tract to initiate infection in
lymphoid tissue before invasion of the central nervous system. Yet the
plausibility of CWD spreading to people has remained elusive.
Part of the problem seems to stem from the US surveillance system. CJD
is only reported in those areas known to be endemic foci of CWD.
Moreover, US authorities have been criticised for not having performed
enough prionic tests in farm deer and elk.
Although in November last year the US Food and Drug Administration
issued a directive to state public-health and agriculture officials
prohibiting material from CWD-positive animals from being used as an
ingredient in feed for any animal species, epidemiological control and
research in the USA has been quite different from the situation in the
UK and Europe regarding BSE.
Getting data on TSEs in the USA from the government is like pulling
teeth, Singeltary argues. You get it when they want you to have it,
and only what they want you to have.
Norman Foster, director of the Cognitive Disorders Clinic at the
University of Michigan (Ann Arbor, MI, USA), says that current
surveillance of prion disease in people in the USA is inadequate to
detect whether CWD is occurring in human beings; adding that, the
cases that we know about are reassuring, because they do not suggest the
appearance of a new variant of CJD in the USA or atypical features in
patients that might be exposed to CWD. However, until we establish a
system that identifies and analyses a high proportion of suspected prion
disease cases we will not know for sure. The USA should develop a
system modelled on that established in the UK, he points out.
Ali Samii, a neurologist at Seattle VA Medical Center who recently
reported the cases of three hunters two of whom were friends who died
from pathologically confirmed CJD, says that at present there are
insufficient data to claim transmission of CWD into humans; adding that
[only] by asking [the questions of venison consumption and deer/elk
hunting] in every case can we collect suspect cases and look into the
plausibility of transmission further. Samii argues that by making both
doctors and hunters more aware of the possibility of prions spreading
through eating venison, doctors treating hunters with dementia can
consider a possible prion disease, and doctors treating CJD patients
will know to ask whether they ate venison.
CDC spokesman Ermias Belay says that the CDC will not be investigating
the [Samii] cases because there is no evidence that the men ate
CWD-infected meat. He notes that although the likelihood of CWD
jumping the species barrier to infect humans cannot be ruled out 100%
and that [we] cannot be 100% sure that CWD does not exist in humans&
the data seeking evidence of CWD transmission to humans have been very
limited.
http://infection.thelancet.com/journal/journal.isa
UPDATE, ATYPICAL SCRAPIE NOR98 DOCUMENTED IN USA ;
SCRAPIE UPDATE USA AS OF MARCH 2007 NOR98 INCLUDED
http://www.aphis.usda.gov/animal_health/animal_diseases/scrapie/downloads/monthly_scrapie_rpt.pps
NOR98-LIKE STRAIN OF SCRAPIE FOUND IN WYOMING (1791 lines)
From: Terry S. Singeltary Sr.
Date: Wed, 11 Apr 2007 15:08:15 -0500
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=8315
THE SEVEN SCIENTIST REPORT ***
http://www.fda.gov/ohrms/dockets/dockets/02n0273/02n-0273-EC244-Attach-1.pdf
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE)
http://www.fsis.usda.gov/OPPDE/Comments/2006-0011/2006-0011-1.pdf
[Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk
Materials for Human Food and Requirement for the Disposition of
Non-Ambulatory Disabled Cattle
03-025IFA
03-025IFA-2
Terry S. Singeltary
9/13/2005
http://www.fsis.usda.gov/OPPDE/Comments/03-025IFA/03-025IFA-2.pdf
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
http://www.chron.com/disp/story.mpl/ap/tx/4797528.html
