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From: TSS ()
Subject: Birth cohort of CANADIAN BSE-positive animal was exported to the United States
Date: April 10, 2007 at 7:58 am PST

Beef News
Birth cohort of BSE-positive animal was exported to the United States

By John Gregerson on 4/10/2007 for

One of the birth cohorts of a Canadian bull diagnosed with bovine spongiform encephalopathy in January was exported to the United States in 2002, according to USDA's Animal and Plant Health Inspection Service.

The animal, a heifer, was sent to a Nebraska feedlot, and then was slaughtered at a Nebraska facility. APHIS indicated the animal presented a negligible risk since scientific data indicate that two BSE-positive animals rarely originate from the same herd. "Even at the height of the BSE epidemic in Britain, it was extremely rare to find a cohort at risk for the disease," APHIS spokeswoman Andrea McNally told

APHIS spokeswoman Karen Eggerg added that the data on cohorts is based on "years of observation" rather than clinical studies, and indicated that one theory why two animals from the same herd are rarely BSE-positive is that prions, the misfolded proteins associated with BSE, generally are clumped together as a result of their sticky nature, and therefore aren't evenly distributed in feed.

After discovering the infected bull, a 79-month-old animal from Alberta, the Canadian Food Inspection Agency focused on cattle born in the same herd within 12 months. The bull became emaciated last winter and subsequently was earmarked for Canada's National BSE Surveillance Program.

funny, i must be slipping, i did not see this on any of the usda/aphis updates.
must have another bse/base mad cow website somewhere?
i'm still waiting for official annoucnement of how safe we are from those nor98 TSE now documented in the USA too, nothing there yet either???
wonder why old ron or johanns have not come out and stated how safe we are yet from any exported mad cows from canada ???
i'm sure this is just a matter of overlook, as to we all know how USDA/APHIS et al BSE testing and reporting is done in such a timely manner, 4 to 8 months after the fact......tss



39. Professor John Wilesmith (Defra) updated the committee on the

BSE cases born after the 1996 reinforced mammalian meat and

bone meal ban in the UK (BARB cases). Around 116 BARB cases

had been identified in Great Britain up to 22 November 2005,

mostly through active surveillance. BARB cases had decreased in

successive birth cohorts, from 44 in the 1996/1997 cohort to none

to date in the 2000/2001 cohort. However, 3 BARB cases had

been identified in the 2001/2002 cohort. Backcalculation of the

prevalence of BARB cases indicated a drop from 130 infected

animals per million (95% confidence interval 90-190) in the

1996/1997 cohort to 30 infected animals per million (95%

confidence interval 10-60) in the 1999/2000 cohort. A shift in the

geographical distribution of BSE cases, from the concentration of

pre-1996 BSE cases in Eastern England to a more uniform


© SEAC 2005

distribution of BARB cases, had occurred. However, it appeared

that certain post-1996 cohorts had a higher exposure to BSE in

certain areas for limited periods. Several clusters of BARB cases

within herds had been identified (5 pairs, 2 triplets and 1


40. A triplet of BARB cases in South West Wales had been

investigated in detail. The triplet comprised 2 cases born in

September and October 2001 and a third in May 2002. The

animals born in 2001 were reared outdoors from the spring of 2002

but the animal born in 2002 had been reared indoors. Further

investigation of feeding practices revealed that a new feed bin for

the adult dairy herd had been installed in September 1998. In July

2002 the feed bin was emptied, but not cleaned, and relocated. All

3 BARB cases received feed from the relocated bin. This finding

suggested the hypothesis that the feed bin installed in September

1998 was filled initially with contaminated feed, that remnants of

this feed fell to the bottom of the bin during its relocation, and thus

young animals in the 2001/2002 birth cohort were exposed to

feedstuffs produced in 1998. No adult cattle had been infected

because of the reduced susceptibility to BSE with increasing age.

41. Further investigation of multiple case herds had found no

association of BARB clusters with the closure of feed mills.

42. Professor Wilesmith concluded that there is evidence of a decline

in risk of infection for successive birth cohorts of cattle. The BARB

epidemic is unlikely to be sustained by animals born after 31 July

2000. Feed bins could represent a continued source of occasional

infection and advice to farmers is being formulated to reduce this

risk. There is no evidence for an indigenous source of infection for

the BARB cases.

43. Members considered it encouraging that no other factor, apart from

feed contamination, had been identified as a possible cause of

BARB cases to date. Members commented that this study

suggests that only a small amount of contaminated feed may be

required for infection and that BSE infectivity can survive in the

environment for several years. Professor Wilesmith agreed and

noted that infection caused by small doses of infectious material

was consistent with other studies, and it would appear there is little

dilution of infectivity, if present, in the rendering system.

Additionally it appeared that the infectious agent had survived for 4

years in the feed bin.

44. The Chair thanked Professor Wilesmith for his presentation.


23.2 BSE-infected mad cows in the standing Canadian adult cattle population. very disturbing...


BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01

Importation of Certain Commodities From BSE Minimal-risk Regions (Canada)

Environmental Assessment, October 27, 2006



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