SEARCH VEGSOURCE:

 

 

Follow Ups | Post Followup | Back to Discussion Board | VegSource
See spam or
inappropriate posts?
Please let us know.
  




From: TSS ()
Subject: UK's first case of H-type BSE confirmed by DEFRA
Date: March 9, 2007 at 11:03 am PST

UK's first case of H-type BSE confirmed by DEFRA

09/03/2007 16:28:00
FWi
DEFRA has reported the UKís first case of H-type Bovine Spongiform Encephalopathy. During routine research carried out by the Veterinary Laboratories Agency, test results revealed a single case of H-type BSE in 2005.

News of this form of BSE, which produces no clinical signs and has only been found in older animals, was announced at a VLA symposium on prion diseases of domestic livestock in June 2006.


To date 18 cases of the H-type strain have been identified in 12 countries worldwide including several other European countries, Japan, Canada and the United States of America.

According to statement from DEFRA the cow died on a farm in Dumfries and Galloway at 13 years of age. The carcass was then sampled and tested for BSE in accordance with EU requirements for testing all fallen stock aged over 24 months.

Test results confirmed the presence of BSE. The carcass was incinerated and the offspring from this cow were culled in line with current BSE regulations.

In August 2006 the VLA began a retrospective examination of brain samples taken from previous BSE cases as part of a research programme designed to analyse different types of BSE in the national herd.


It was this exercise that identified the presence of a H-type case. All forms of BSE are treated identically therefore no additional action would have been required in this case.

The Spongiform Encephalopathy Advisory Committee will examine all the available data on this case during a discussion of different forms of BSE worldwide at their next meeting on 10 May 2007.

by Andrew Watts (About this Author)

http://www.fwi.co.uk/Articles/2007/03/09/102241/uks-first-case-of-h-type-bse-confirmed-by-defra.html

U.K. TSE RESEARCH 2005-2008

http://www.defra.gov.uk/animalh/Bse/pdf/tse-strategy0606.pdf

BSE cases in older cattle which resemble the two different atypical. BSE phenotypes that have recently been described in France. (designated H-type) and ...


http://www.seac.gov.uk/papers/96-2.pdf

----- Original Message -----
From: XXXXXXXX XXXXXXXXXXXX(SEAC)
To: 'flounder9@verizon.net'
Sent: Friday, February 23, 2007 8:13 AM
Subject: RE: SEAC ANNUAL REPORT 2006


Dear Mr Singeltary

Thank you for your email about a possible relationship between BASE and
sCJD.

SEAC is continually informed about and considers the emerging science on
both animal and human TSEs. The committee regularly receives updates from
the UK's National CJD Surveillance Centre Unit on the epidemiology of vCJD
and sCJD and is also aware of emerging research on BASE. It is envisaged
that SEAC will conduct a detailed consideration of the available science and
the possible animal and human health implications of BASE at its meeting on
10th May 2007.

Yours sincerely

XXXXXXXXXXXXXX


----------------------------------------------------------------------------
----
From: Terry S. Singeltary Sr. [mailto:flounder9@verizon.net]
Sent: 02 February 2007 16:44
To: Bovine Spongiform Encephalopathy
Cc: SEACsecretariat (AHEG); cjdvoice@yahoogroups.com;
BLOODCJD@YAHOOGROUPS.COM; madcow@lists.iatp.org; Sustainable Agriculture
Network Discussion Group
Subject: SEAC ANNUAL REPORT 2006


Date: February 2, 2007 at 8:25 am PST
SEAC ANNUAL REPORT 2006 (ignoring the obvious)


http://www.seac.gov.uk/publicats/annualreport2006.pdf


i guess the sporadic CJD cases have all be resolved, and the route and cause
classified.
to this day, i cannot figure this out. if BSE farmers died from sporadic
CJD, and now another study seems to point
that BSE and BASE may be the same thing, and that BASE does not produce vCJD
like pathology, but pathology resembling that of sporadic CJD, then why does
SEAC still seem to put sporadic CJD on the back burners ??? IN fact, SEAC
could not even speak about it in there 2006 report. would it not seem
prudent to mention these findings in this 2006 SEAC report ;


Other work presented suggested that BSE and bovine amyloidotic spongiform
encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the
prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A
MUTATION FOUND IN CASES OF SPORADIC CJD.


snip...


http://www.seac.gov.uk/minutes/95.pdf


***These results indicate that BASE is transmissible to humans and suggest
that BASE is more virulent than
classical BSE in humans.***


6:30 Close of Day One


http://www.healthtech.com/2007/tse/day1.asp


64. A member noted that at the recent Neuroprion meeting, a study was
presented showing that in transgenic mice BSE passaged in sheep may be more
virulent and infectious to a wider range of species than bovine derived BSE.


http://www.seac.gov.uk/minutes/95.pdf


Asante/Collinge et al, that BSE transmission to the 129-methionine

genotype can lead to an alternate phenotype that is indistinguishable

from type 2 PrPSc, the commonest _sporadic_ CJD;


http://www.fda.gov/ohrms/dockets/ac/03/slides/3923s1_OPH.htm


I STILL think to ignore these findings below is not only rediculous, but
indeed very suspicious ;


CJD FARMERS WIFE 1989


http://www.bseinquiry.gov.uk/files/yb/1989/10/13007001.pdf


http://www.bseinquiry.gov.uk/files/yb/1989/10/13003001.pdf

cover-up of 4th farm worker ???


http://www.bseinquiry.gov.uk/files/yb/1995/10/23006001.pdf


http://www.bseinquiry.gov.uk/files/yb/1995/10/20006001.pdf


CONFIRMATION OF CJD IN FOURTH FARMER


http://www.bseinquiry.gov.uk/files/yb/1995/11/03008001.pdf


now story changes from;

SEAC concluded that, if the fourth case were confirmed, it would be
worrying, especially as all four farmers with CJD would have had BSE
cases on their farms.

to;

This is not unexpected...

was another farmer expected?

http://www.bseinquiry.gov.uk/files/yb/1995/11/13010001.pdf


4th farmer, and 1st teenager

http://www.bseinquiry.gov.uk/files/yb/1996/02/27003001.pdf


2. snip...


Over a 5 year period, which is the time period on which the advice
from Professor Smith and Dr. Gore was based, and assuming a
population of 120,000 dairy farm workers, and an annual incidence
of 1 per million cases of CJD in the general population, a
DAIRY FARM WORKER IS 5 TIMES MORE LIKELY THAN
an individual in the general population to develop CJD. Using the
actual current annual incidence of CJD in the UK of 0.7 per
million, this figure becomes 7.5 TIMES.

3. You will recall that the advice provided by Professor Smith in
1993 and by Dr. Gore this month used the sub-population of dairy
farm workers who had had a case of BSE on their farms -
63,000, which is approximately half the number of dairy farm
workers - as a denominator. If the above sums are repeated using
this denominator population, taking an annual incidence in the general
population of 1 per million the observed rate in this sub-population
is 10 TIMES, and taking an annual incidence of 0.7 per million,
IT IS 15 TIMES (THE ''WORST CASE'' SCENARIO) than
that in the general population...


http://www.bseinquiry.gov.uk/files/yb/1995/01/31004001.pdf


CJD YOUNG PEOPLE

in the USA, a 16 year old in 1978;

ALSO IN USA;

(20 year old died from sCJD in USA in 1980 and a 16 year
old in 1981. see second url below)

in France, a 19 year old in 1982;

in Canada, a 14 year old of UK origin in 1988;

in Poland, cases in people aged 19, 23, and 27 were identified in
a retrospective study (published 1991), having been originally
misdiagnosed with a viral encephalitis;

Creutzfeldt's first patient in 1923 was aged 23.

http://www.bseinquiry.gov.uk/files/yb/1995/10/27013001.pdf

20 year old died from sCJD in USA in 1980 and a 16 year
old in 1981. A 19 year old died from sCJD in
France in 1985. There is no evidence of an iatrogenic
cause for those cases....

http://www.bseinquiry.gov.uk/files/yb/1995/10/04004001.pdf


FOR SEAC TO continue to flounder with this ukbsenvcjd only theory, when we
know that sporadic CJD's were proven long ago to transmit via the medical
and surgical arena, will only continue to spread this agent around the globe
via a multitude of proven routes and sources.

IN my opinion, for SEAC to continue to go down this same road, year after
year, and continue to ignore sporadic CJD's, and continue to say that "BSE
in the UK sheep population is most likely to be zero, or very low if present
at all." "Consequently, any impact of the schemes on human health from
removing BSE from sheep is likely to be negligible." when in fact they do
not know. and in fact science is showing that in "transgenic mice BSE
passaged in sheep may be more virulent and infectious to a wider range of
species than bovine derived BSE." IN my opinion this continued neglect of
other TSEs in human and animals, the continued belief in this ukbsenvcjd
only theory, and the risk thereof, is reckless, dangerous, and in my mind,
criminal. SEAC has failed the public terribly in this regard. ...TSS

----- Original Message -----
From: "Terry S. Singeltary Sr."
To:
Sent: Friday, February 02, 2007 10:43 AM
Subject: SEAC ANNUAL REPORT 2006


Date: February 2, 2007 at 8:25 am PST
SEAC ANNUAL REPORT 2006 (ignoring the obvious)


http://www.seac.gov.uk/publicats/annualreport2006.pdf


SOME HISTORY ON THE USA CJD QUESTIONNAIRE

http://www.ask.com/web?q=cjd+questionnaire+tss&qsrc=1&o=0&l=dir

http://www.google.com/search?num=30&hl=en&lr=&as_qdr=d&edition=us&q=cjd+questionnaire+TSS&btnG=Search

One reason for this was the _inaccuracy_ in coding of cases correctly
certified as CJD Coding is carried out by staff who are not medically
qualified and it is not surprising that coding errors occur in the
processing of large numbers of certificates. In 1982, 12,000
certificates per week were processed at the office of population
censuses and surveys bu 15 coders and 6 checkers (Alderson et al., 1983).
The occurrence of both inter- and intra-observer coding errors has been
described (Curb et al., 1983) and the _inaccuracies_ of BOTH
certification and coding discovered in this study _support_ the
introduction of a more accurate system of death certificates and
a more detailed and specific coding system...

snip...

http://www.bseinquiry.gov.uk/files/mb/m26/tab01.pdf

''Answering critics like Terry Singeltary, who feels that the US
undercounts CJD, Schonberger _conceded_ that the current surveillance
system has errors but stated that most of the errors will be confined to
the older population''...

The Pathological Protein: Mad Cow, Chronic Wasting, and Other Deadly
Prion Diseases by Philip Yam

Philip Yam The Pathological Protein Mad Cow, Chronic Wasting, and Other
Deadly Prion Diseases 2003. Hardcover, 285 pp. Euro 29.95 (net price);
√¬£21.00; $27.50; sFr 51.50 ISBN 0-387-95508-9 Contact and review
copies: Joan Robinson Springer-Verlag Press and Public Relations Tel.:
+49- (0) 6221-487-8130, Fax: +49- (0) 6221-487-8141, E-mail:
robinson@springer.de http://www.springer.de/press/newbooks/protein.html
The Pathological Protein: Mad Cow, Chronic Wasting, and Other Deadly
Prion Diseases Philip Yam List Price: $27.50 Our Price: $19.25 You Save:
$8.25 (30%) Availability: Usually ships within 24 hours.

http://www.target.com/gp/detail.html/sr=1-1/qid=1054420048/ref=sr_1_1/602-9780634-9614260?asin=0387955089

SAD THAT OLDER FOLKS SEEM TO BE EXPENDABLE IN RELATIONS TO TSEs.

IF YOUR NOT YOUNG, IF IT's NOT THE UKBSENVCJD ONLY STRAIN, YOUR OUTA LUCK.

YOUR SPORADIC, YOUR SPONTANEOUS, in short, your expendable. ...

> Number of BSE cases in the EU-25 down again by more than 40 percent in 2006.

> This decreasing trend now seems to occur throughout the EU-25

http://www.fas.usda.gov/gainfiles/200703/146280315.pdf

SEEMS the drastic reduction in BSE cases in the EU due to feed ban would dispute the spontaneous event for BSE, so what do they try and do, convince everyone there is a new strain that happens spontaneously, what a hoot. why not a new strain via the same damn route and source i.e. tainted feed just different strain of agent in feed, or maybe vertical and or horizontal route and or source. but to just throw our hands up in the air and say "OH, it just happens on it's on due to some whaco event of a spontanoeus good protein misfolding into a bad one'' and then replicating themselves into more bad apples, just because we find a new strain of mad cow in the bovine that IS related to sporadic CJDs, flies in the face of past science. it does not compute. and if you go back to those mad cow farmers and one wife, with BSE that died from sporadic CJD, now that does compute.

lets do some pondering here, lets say just for BSe, lets just say this BASE is spontaneous, would this not be the final gold straw in prusiners pie. would every country out there then be subject to 100% testing if say this spontaneous theory was proven, and shoot his cdi testing up big time, and others $$$ holy mad cow, just think about it, over 20+ strains of scrapie, atypical scrapies, BSE in sheep and goat, holy smokes, then you have the multiple strains of CWD they have just about finally confirmed of deer and elk, but how many strains there, alot of testing would have to be done if ever proven to be a spontaneous event. but how can it be spontaneous in one and not the others $$$

i have ask prusiner this very thing, with no answer. how many of the 85%+ of all human TSE i.e. sporadic CJD, how many do you believe to be a spontaneous event, from no route and source, knowing that we have to date 6 different phenotypes of sporadic CJD ??? what, 2%, 10%, 25%, 50%, 75%, or all of the sporadic CJDs spontaneous ??? no explanation yet from him, but i'm still waiting. ...

see Prusiner ;

http://www.agobservatory.org/library.cfm?refID=30405

see singeltary question to prusiner ;

Greetings,as a lay person;-) i am thankful for Dr. Prusiners report below. I only wish that he would elaborate on the spontaneous aspect of sporadic CJD and how many of the 85%+ of all CJDs does he think happens spontaneously without route and source of the agent? I am concerned that people who read this, will come to the conclusion that all sporadic CJDs are a spontaneousmutation, when in reality all sporadic CJD is, is CJD from unknown route and source and they could be many. in fact, there could be many phenotypes of CJD that are now called sporadic CJD...

http://www.agobservatory.org/library.cfm?refID=30406


TSS





Follow Ups:



Post a Followup

Name:
E-mail: (optional)
Subject:

Comments:

Optional Link URL:
Link Title:
Optional Image URL: