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From: TSS ()
In Reply to: Pennsylvania Game Commission Law and Law Makers Message Board HUNTING PA.COM BANS SPEAKING ABOUT CWD posted by TSS on December 21, 2005 at 8:37 am:
We cannot proceed. Please use your back button to return to the previous page. ----- Original Message ----- > By the way, Joe Neville said he will see if he can find out why your info i still cannot figure it out. i have posted nothing obscene, abusive, just kind regards, ----- Original Message ----- -----Original Message----- whether you know about this or not, or even care. i have done nothing wrong kind regards, Subject: Pennsylvania Game Commission Law and Law Makers Message Board Greetings State of PA hunters et al, SEEMS Big Brother is working everywhere these days. after 8 years of trying http://www.huntingpa.com/ubbthreads/ubbthreads.php?Cat= Law and Law Makers Message Board >> Pennsylvania Game Commission GAME COMMISSION BANS IMPORTATION OF CERTAIN CARCASS PARTS FROM STATES WITH Using newly-granted emergency powers, Pennsylvania Game Commission Executive The ban closely mirrors a similar ban issued on Sept. 21 by the state "With chronic wasting disease (CWD) present in free-ranging and captive "There are many scientific unknowns about CWD, so this order may need to be For more information, please visit the Pennsylvania Game Commission - State Greetings Samuel and Hunters of PA, i will no longer be able to post here for reasons posted below. sorry for From: Samuel Terry, Thank you for your concern in this matter and keeping a close vigil on the Samuel I have been ask not to post anymore here about CWD. Like I had asked you, please refrain from posting information on CWD. Thank you. Pennsylvania Game Commission Law and Law Makers Message Board HUNTING PA.COM TSS ============================== New Mexico Department of Game and Fish FOR IMMEDIATE RELEASE, DEC. 9, 2005: TWO NEW MEXICO ELK TEST POSITIVE FOR CHRONIC WASTING DISEASE SANTA FE - Two elk killed in the southern Sacramento Mountains of southeast Both CWD-afflicted elk were killed in an area 10 to 15 miles southeast of "The range in which the disease is found appears to be expanding, so finding Brain stem samples from the two elk were among more than 100 taken from deer "We will continue our efforts to monitor the disease by actively testing This season, hunters who kill animals in a "Control Area" of Unit 34 are Chronic wasting disease is a fatal neurological illness that afflicts deer, The origin of CWD in New Mexico is unknown. It has been found in 12 wild For more information about CWD in New Mexico, including special regulations ### http://www.wildlife.state.nm.us/publications/press_releases/documents/10-9cw CWD CONTROL MAP NM http://www.wildlife.state.nm.us/documents/cwdcontrolmap.pdf #################### https://lists.aegee.org/bse-l.html Sponsored in part by Elsevier, publisher of [The UK Department of Health website has been revised and the monthly Data on vCJD cases from all parts of the world are now included in In this update: [1] UK: DH vCJD Monthly Statistics - as of Fri 4 Nov 2005 ***** Definite and probable CJD cases in the UK: Summary of vCJD Cases - Deaths Deaths from definite vCJD (confirmed): 108 Deaths from probable vCJD (without neuropathological confirmation): 43 Deaths from probable vCJD (neuropathological confirmation pending): 1 Number of deaths from definite or probable vCJD (as above): 152 Summary of vCJD Cases - Alive Number of probable vCJD cases still alive: 6 Total Number of definite or probable vCJD (dead and alive): 158 (The next table will be published on Mon 5 Dec 2005) [Since the previous monthly statistics were released on Fri 30 Sep These data are consistent with the view that the vCJD outbreak in the -- [As of 30 Sep 2005, so far in the UK for the year 2005 there have I am indebted to Terry S. Singeltary Sr. Since 1997 in the USA there have been 876 confirmed cases of sporadic The corresponding totals for confirmed diagnoses in the UK since 1990 ****** Background Recently a further hypothesis has been considered by SEAC, which Contamination of animal feed with human remains 4. It is likely that similar material was imported into other Properties of BSE and other TSEs 6. Prion strains can be characterised by their properties in 7. Nevertheless it is difficult, and often impossible with current Summary of SEAC's discussion 9. It is not possible to determine, from current knowledge of the 10. There was evidence to suggest that human remains may have been 11. Very much larger quantities of cattle and sheep remains 12. In conclusion, although Professor Colchester's hypothesis can 13. Although further experiments suggested by Professor Colchester -- ******* (1) Virologie Immunologie Moleculaires and (2) Genetique Biochimique The Abstract of the paper reads as follows: "Scrapie in small -- [These findings need to be considered in the context of current ****** The Abstract reads as follows: "Chronic wasting disease (CWD) is an -- ****** Adriano Aguzzi, the lead researcher on the study, has not detected Prions are mainly found in the brain, spinal cord and immune system. The researchers went to Sardinia, a Mediterranean island with more The prion concentration in the sheep's mammary glands is thousands of [Byline: Andreas von Bubnoff] -- [see also: CJD (new var.) - UK: update Jan 2002 20020111.3223 *##########################################################* [Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk 03-025IFA From: Terry S. Singeltary Sr. [flounder9@verizon.net] Sent: Thursday, September 08, 2005 6:17 PM To: fsis.regulationscomments@fsis.usda.gov Subject: [Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified for the Disposition of Non-Ambulatory Disabled Cattle Greetings FSIS, I would kindly like to submit the following to [Docket No. 03-025IFA] FSIS Requirements for the Disposition of Non-Ambulatory Disabled Cattle THE BSE/TSE SUB CLINICAL Non-Ambulatory Disabled Cattle Broken bones and such may be the first signs of a sub clinical BSE/TSE snip...FULL TEXT ; http://docket.epa.gov/edkfed/do/EDKStaffAttachDownloadPDF?objectId=090007d48 http://docket.epa.gov/edkfed/do/EDKStaffCollectionDetailView?objectId=0b0007 ======================================================== ======================================================== OLD TSS SUBMISSIONS; Docket No, 04-047-l Regulatory Identification No. (RIN) 091O-AF46 NEW BSE https://web01.aphis.usda.gov/regpublic.nsf/0/eff9eff1f7c5cf2b87256ecf000df08 http://www.fda.gov/ohrms/dockets/dockets/03n0312/03N-0312_emc-000001.txt Docket Management Docket: 02N-0273 - Substances Prohibited From Use in Animal Food or Feed; Animal Proteins Prohibited in Ruminant Feed Comment Number: EC -10 Accepted - Volume 2 PART 2 PDF]Freas, William TSS SUBMISSION File Format: PDF/Adobe Acrobat - Page 1. J Freas, William From: Sent: To: Subject: Terry S. Singeltary Sr. [flounder@wt.net] Monday, January 08,200l 3:03 PM freas ... http://www.fda.gov/ohrms/dockets/ac/01/slides/3681s2_09.pdf Asante/Collinge et al, that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest _sporadic_ CJD; http://www.fda.gov/ohrms/dockets/ac/03/slides/3923s1_OPH.htm Docket Management Docket: 96N-0417 - Current Good Manufacturing Practice http://www.fda.gov/ohrms/dockets/dailys/03/Mar03/031403/96N-0417-EC-2.htm Docket No. 2003N-0312 Animal Feed Safety System [TSS SUBMISSION http://www.fda.gov/ohrms/dockets/dockets/03n0312/03N-0312_emc-000001.txt # Docket No: 02-088-1 RE-Agricultural Bioterrorism Protection Act of Docket Management Docket: 02N-0276 - Bioterrorism Preparedness; Registration of Food http://www.fda.gov/ohrms/dockets/dockets/02n0276/02N-0276-EC-254.htm OTC External Analgesic Drug Products, ... EMC 7, Terry S. Singeltary Sr. http://www.fda.gov/ohrms/dockets/dailys/03/oct03/100203/100203.htm DOCKETS ENTERED on 2/5/03. ... EMC 4 Terry S. Singeltary Sr. Vol#: 2. 03N-0009 Federal Preemption of State & Local Medical Device Requireme. ... Docket: 02N-0370 - Neurological Devices; Classification of Human Dura Mater Comment Number: EC -1 Accepted - Volume 1 ... 00D-1662 Use of Xenotransplantation Products in Humans. http://www.fda.gov/ohrms/dockets/dailys/03/Jun03/060903/060903.htm 2003D-0186 01N-0423 Substances Prohibited from use in animal food/Feed Ruminant APE 5 National Renderers Association, Inc. Vol#: 2 APE 6 Animal Protein Producers Industry Vol#: 2 APE 7 Darling International Inc. Vol#: 2 EMC 1 Terry S. Singeltary Sr. Vol#: 3 http://www.fda.gov/ohrms/dockets/dailys/01/Oct01/101501/101501.htm Send Post-Publication Peer Review to journal: disease in the United States I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc? LANCET INFECTIOUS DISEASE JOURNAL My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem. largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD)the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation. occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally , he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source. region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle. transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive. is only reported in those areas known to be endemic foci of CWD. Moreover, US authorities have been criticised for not having performed enough prionic tests in farm deer and elk. issued a directive to state public-health and agriculture officials prohibiting material from CWD-positive animals from being used as an ingredient in feed for any animal species, epidemiological control and research in the USA has been quite different from the situation in the UK and Europe regarding BSE. teeth , Singeltary argues. You get it when they want you to have it, and only what they want you to have. University of Michigan (Ann Arbor, MI, USA), says that current surveillance of prion disease in people in the USA is inadequate to detect whether CWD is occurring in human beings ; adding that, the cases that we know about are reassuring, because they do not suggest the appearance of a new variant of CJD in the USA or atypical features in patients that might be exposed to CWD. However, until we establish a system that identifies and analyses a high proportion of suspected prion disease cases we will not know for sure . The USA should develop a system modelled on that established in the UK, he points out. Ali Samii, a neurologist at Seattle VA Medical Center who recently reported the cases of three hunterstwo of whom were friendswho died from pathologically confirmed CJD, says that at present there are insufficient data to claim transmission of CWD into humans ; adding that [only] by asking [the questions of venison consumption and deer/elk hunting] in every case can we collect suspect cases and look into the plausibility of transmission further . Samii argues that by making both doctors and hunters more aware of the possibility of prions spreading through eating venison, doctors treating hunters with dementia can consider a possible prion disease, and doctors treating CJD patients will know to ask whether they ate venison. the [Samii] cases because there is no evidence that the men ate CWD-infected meat . He notes that although the likelihood of CWD jumping the species barrier to infect humans cannot be ruled out 100% and that [we] cannot be 100% sure that CWD does not exist in humans& the data seeking evidence of CWD transmission to humans have been very limited . Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source. <<< actually, that quote was from a more recent article in the Journal of Neurology (see below), not the JAMA article... Full Text Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits= BRITISH MEDICAL JOURNAL SOMETHING TO CHEW ON BMJ http://www.bmj.com/cgi/eletters/319/7220/1312/b#EL2 BMJ http://www.bmj.com/cgi/eletters/320/7226/8/b#EL1 THE PATHOLOGICAL PROTEIN BY Philip Yam Yam Philip Yam News Editor Scientific American www.sciam.com IN light of Asante/Collinge et al findings that BSE transmission to the -------- Original Message -------- Subject: re-BSE prions propagate as either variant CJD-like or sporadic CJD Date: Thu, 28 Nov 2002 10:23:43 -0000 From: "Asante, Emmanuel A" To: Dear Terry, I have been asked by Professor Collinge to respond to your request. I am a Senior Scientist in the MRC Prion Unit and the lead author on the paper. I have attached a pdf copy of the paper for your attention. Thank you for your interest in the paper. In respect of your first question, the simple answer is, yes. As you will find in the paper, we have managed to associate the alternate phenotype to type 2 PrPSc, the commonest sporadic CJD. It is too early to be able to claim any further sub-classification in respect of Heidenhain variant CJD or Vicky Rimmer's version. It will take further studies, which are on-going, to establish if there are sub-types to our initial finding which we are now reporting. The main point of the paper is that, as well as leading to the expected new variant CJD phenotype, BSE transmission to the 129-methionine genotype can lead to an alternate phenotype which is indistinguishable from type 2 PrPSc. I hope reading the paper will enlighten you more on the subject. If I can be of any further assistance please to not hesitate to ask. Best wishes. Emmanuel Asante <> ____________________________________ Dr. Emmanuel A Asante MRC Prion Unit & Neurogenetics Dept. Imperial College School of Medicine (St. Mary's) Norfolk Place, LONDON W2 1PG Tel: +44 (0)20 7594 3794 Fax: +44 (0)20 7706 3272 email: e.asante@ic.ac.uk (until 9/12/02) New e-mail: e.asante@prion.ucl.ac.uk (active from now) ____________________________________ snip... full text ; http://www.fda.gov/ohrms/dockets/ac/03/slides/3923s1_OPH.htm Creutzfeldt-Jakob disease THE findings from Corinne Ida Lasmézas*, [dagger] , Jean-Guy Fournier*, Hermann Boe*, Domíníque Marcé*, François Lamoury*, Nicolas Kopp [Dagger ] , Jean-Jacques Hauw§, James Ironside¶, Moira Bruce [||] , Dominique Dormont*, and Jean-Philippe Deslys* et al, that The agent responsible http://www.pnas.org/cgi/content/full/041490898v1 Characterization of two distinct prion strains http://vir.sgmjournals.org/cgi/content/abstract/85/8/2471 ALL human TSEs must be made reportable Nationally and Internationally, OF
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