Here are exerpts from studies re the effects of meat on human digestion. Bottom line meat is toxic to humans. Try eating nothing but meat for a week and see how you feel. Then try eating nothing but fresh fruits and veggies for a week and see how you feel. Therin lies the answer.

J Nutr 1975 Jul;105(7):878-84
Effects of high risk and low risk diets for colon carcinogenesis on fecal microflora and steroids in man.
Reddy BS, Weisburger JH, Wynder EL.
We investigated the effects of a high meat mixed Western diet and a nonmeat diet, representing the dietary pattern of high and low risk areas for colon cancer, respectively, on fecal microflora dn on bile acid and neutral sterol patterns in man. The total anaerobic microflora as well as the count of Bacteroides, Bifidobacterium, Peptococcus, and anaerobic Lactobacillus were significantly higher during the period of consumption of a high meat mixed Western diet comparted with the nonmeat-diet consumption period. The difference in total fecal bile acid excretion was not significant between the two dietary periods. Fecal excretion of microbially modified bile acids and neutral sterols was decreased when subjects eating a high meat diet transferred to a nonmeat diet. These results support the fact that diet plays a modifying role on the composition of intestinal microflora, bile acids, and neutral sterols.

Acta Physiol Hung 1999;86(3-4):171-80
Vegan diet in physiological health promotion.
Hanninen O, Rauma AL, Kaartinen K, Nenonen M. Department of Physiology, University of Kuopio, Finland.
We have performed a number of studies including dietary interventions and cross-sectional studies on subjects consuming uncooked vegan food called living food (LF) and clarified the changes in several parameters related to health risk factors. LF consists of germinated seeds, cereals, sprouts, vegetables, fruits, berries and nuts. Some items are fermented and contain a lot of lactobacilli. The diet is rich in fiber. It has very little sodium, and it contains no cholesterol. Food items like berries and wheat grass juice are rich in antioxidants such as carotenoids and flavonoids. The subjects eating living food show increased levels of carotenoids and vitamins C and E and lowered cholesterol concentration in their sera. Urinary excretion of sodium is only a fraction of the omnivorous controls. Also urinary output of phenol and p-cresol is lowered as are several fecal enzyme levels which are considered harmful. The rheumatoid arthritis patients eating the LF diet reported amelioration of their pain, swelling of joints and morning stiffness which all got worse after finishing LF diet. The composite indices of objective measures showed also improvement of the rheumatoid arthritis patients during the intervention. The fibromyalgic subjects eating LF lost weight compared to their omnivorous controls. The results on their joint stiffness and pain (visual analogue scale), on their quality of sleep, on health assessment questionnaire and on general health questionnaire all improved. It appears that the adoption of vegan diet exemplified by the living food leads to a lessening of several health risk factors to cardiovascular diseases and cancer. Rheumatoid patients subjectively benefited from the vegan diet which was also seen in serum parameters and fecal analyses.

F Kassie, EF Lhoste, A Bruneau, M Zsivkovits, F Ferk, M Uhl, T Zidek, and S Knasmuller
Effect of intestinal microfloras from vegetarians and meat eaters on the genotoxicity of 2-amino-3-methylimidazo[4,5-f]quinoline, a carcinogenic heterocyclic amine.
J Chromatogr B Analyt Technol Biomed Life Sci, March 25, 2004; 802(1): 211-5.
Institute of Cancer Research, University of Vienna, Borschkegasse 8A A-1090 Vienna, Austria.
Aim of this study was to investigate the impact of intestinal microfloras from vegetarians and non-vegetarians on the DNA-damaging activity of 2-amino-3-methyl-3H-imidazo[4,5-f]quinoline (IQ), a carcinogenic heterocyclic amine that is found in fried meats. Floras from four vegetarians (Seventh Day Adventists [who tend not to be vegan - ljf]) and from four individuals who consumed high amounts of meats were collected and inoculated into germfree F344 rats. The rats were kept on isocaloric diets that either contained animal derived protein and fat (meat consumers group) or proteins and fat of plant origin (vegetarian groups). IQ (90 mg/kg bw) was administered orally, after 4 h the extent of DNA-damage in colon and liver cells was determined in single cell gel electrophoresis assays. In all groups, the IQ induced DNA-migration was in the liver substantially higher than in the colon. In animals harbouring floras of vegetarians, the extent of damage was in both organs significantly (69.2% in the liver, P

Am J Clin Nutr 2000 Dec;72(6):1488-94
Contribution of dietary protein to sulfide production in the large intestine: an in vitro and a controlled feeding study in humans.
Magee EA, Richardson CJ, Hughes R, Cummings JH. Dunn Clinical Nutrition Centre, Hills Road, Cambridge, United Kingdom. e.magee@dundee.ac.uk
BACKGROUND: Hydrogen sulfide is a luminally acting, bacterially derived cell poison that has been implicated in ulcerative colitis. Sulfide generation in the colon is probably driven by dietary components such as sulfur-containing amino acids (SAAs) and inorganic sulfur (eg, sulfite).
OBJECTIVE: We assessed the contribution of SAAs from meat to sulfide production by intestinal bacteria with use of both a model culture system in vitro and an in vivo human feeding study.
DESIGN: Five healthy men were housed in a metabolic suite and fed a sequence of 5 diets for 10 d each. Meat intake ranged from 0 g/d with a vegetarian diet to 600 g/d with a high-meat diet. Fecal sulfide and urinary sulfate were measured in samples collected on days 9 and 10 of each diet period. Additionally, 5 or 10 g bovine serum albumin or casein/L was added to batch cultures inoculated with feces from 4 healthy volunteers. Concentrations of sulfide, ammonia, and Lowry-reactive substances were measured over 48 h.
RESULTS: Mean (+/-SEM) fecal sulfide concentrations ranged from 0.22 +/- 0.02 mmol/kg with the 0-g/d diet to 3.38 +/- 0.31 mmol/kg with the 600-g/d diet and were significantly related to meat intake (P: CONCLUSION: Dietary protein from meat is an important substrate for sulfide generation by bacteria in the human large intestine.

Possible associations of dietary residues with growth of the large gut. Am. J. Physiol. 197, 903-911).... Discussion: Fiber in the diet seemed to have a growth enhancing effect on the small intestine.

The decreased fecal volume 80-120 grams/day of Western Man and prolonged intestinal transit time of over 72 hours compared to the 300-500 grams/day fecal volume and under 40 hours transit time of the Third World countries, is obviously diet/fiber related. The increased intestinal retention of the waste products of putrefaction, from meats and animal fats, with prolonged exposure of the intestinal mucosa to their toxins and carcinogens is responsible for the emergence and prevalence of many disease states.
Burkitt, D.P. et al. Dietary Fiber and disease. Journal of the American Medical Association. 229:1068, 1974

Red and white meat increased colonocyte DNA single-strand breaks (SS and double-strand breaks (DSdose dependently. Resistant starch (RS), a dietary fibre component, prevented this damage. Apoptotic cells were increased dose dependently by red meat irrespective of RS. Red meat induced greater colonic mucus layer thinning than white meat. Dietary RS protects against this damage and against loss of the mucus barrier through increased butyrate production. Carcinogenesis. 2007 Nov;28(11):2355-62.

Cancer Biol Ther. 2006 Mar;5(3):267-74.
High dietary protein result in increased colonic DNA damage by thinning of colonic mucus layer. Dietary casein caused a 2-fold increase in colonic DNA damage and reduced thickness of colonic mucus layer by 41%. Cooked meat caused 26% greater DNA damage than casein. Cecal and fecal short chain fatty acid pools were increased by inclusion of resistant starch, RS. DNA damage due to high protein could increase colorectal cancer but RS could reduce the risk.

Proc Nutr Soc. 2003 Feb;62(1):31-6.
Colonic diverticula.
Diverticula are a herniation through the wall of the sigmoid colon and are likely to be a consequence of a weakness in the colonic wall or prolonged exposure to increased intracolonic pressure consequent on a low dietary fibre intake. Adult Africans living in Africa eat a high-fibre diet and are free from diverticulosis. Their colons are stronger, wider and thinner than those Scottish adults of the same age.

Undigested protein is fermented by the colonic microflora with the resultant end-products of SCFAs, branched-chain fatty acids (e.g.,isovalerate, isobutyrate, and 2-methylbutyrate), and potentially harmful metabolites –ammonia, amines,phenols, sulfide, and indoles. Altern Med Rev 2004;9(2):180-197
"animals harbouring floras of vegetarians, the extent of damage was in both organs significantly lower than in the meat consumer"J Chromatogr B Analyt Technol Biomed Life Sci, 2004; 802(1): 211-5.

Dietary factors protecting women from urinary tract infection.
Because urinary tract infections (UTIs) are caused by bacteria in the stool, dietary factors may affect the risk of contracting a UTI by altering the properties of the fecal bacterial flora.
Frequent consumption of fresh juices, especially berry juices.. was associated with a decreased risk of recurrence of UTI: the OR for UTI was 0.66 (95% CI: 0.48, 0.92) per 2 dL juice.

As soon as I read the title all I could think was, "Just open your mouth!" Lol. But seriously, I just don't see the appeal in eating something that had to give its life (plants may be alive but in an entirely different manner) just so I can gorge. Thanks for the post!

Thanks for sharing dear buddy keep it up. Everyone has right to say something. So, Everyone should keep it up. And share more ideas Thanks